It’s supposed to be a whole new approach to cholesterol.
It’s hurtling through the approvals process right now, as you read this.
And the moment it gets the FDA rubber-stamp, you can expect to hear a breathless sales pitch from your doctor.
It’s supposed to work better… kick in faster… and pack fewer risks.
But read between the lines, and you’ll notice something really weird about this “new” approach.
It’s full of the same OLD risks!
In fact, once you get the details, you’ll see there’s very little that’s truly “new” about this “new” approach.
Doubling down on cholesterol fighting failure
Don’t get TOO excited about this up-and-coming drug. It’s NOT a replacement for statins, the drugs responsible for countless cases of muscle pain, memory loss, and diabetes.
Instead, you take it WITH your statins.
And this new drug doesn’t work in a new way, either. In fact, it targets the same old pathways as statins.
See what I mean?
But it doesn’t have the same side effects as statins. So they think they can at least cut the statin dose and then ADD this med to the mix. And then BOOM, you get the results without all of the risks.
And in studies, sure, LDL levels plunged.
But here’s the crazy thing: Lower ISN’T automatically better when it comes to LDL cholesterol, and that shouldn’t even be controversial.
It’s a biological fact!
In fact, when LDL sinks too low, you could face big-time problems. One study just last month found women with low cholesterol – levels any conventional doc would be THRILLED to see – have a HIGHER risk of a stroke.
Then there’s another little fact the mainstream doesn’t like to talk about. Three out of four heart attack patients have picture-perfect cholesterol levels.
Inflammation & oxidation are the REAL enemies
So the problem with cholesterol isn’t the number. The number by itself is about as useful as a sixth toe.
The real issue is when cholesterol clogs and hardens your arteries.
Many docs will claim if you’ve got a lot of cholesterol in your arteries, you’ve got too much.
But that’s NOT how it works.
Your arteries are like a superhighway. It can handle the traffic… until there’s an accident. In this case, inflammation in the arteries and oxidation of the LDL are the “accident,” which in turn causes the LDL to get locked in place and stick to the walls.
In other words, it’s NOT the number that matters the most here. It’s the inflammation and oxidation, which can cause backups even when your LDL levels are nice and low.
Instead of cutting LDL, get busy cutting inflammation, with…
- B vitamins
- fish oil
- a diet low in sugar
And of course, fight the oxidation with antioxidants. The job is right in the name, and one of the best for beating back the oxidation that damages LDL cholesterol is astaxanthin.